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[1] LANG AE,LOZANO AM. Parkinson’s disease[J]. Lancet,1998,386(9996):896-912.
[2] PRINGSHEIM T,JETTE N,FROLKIS A,et al. The prevalence of Parkinson’s disease:a systematic review and meta-analysis[J]. Mov Disord,2014,29(13):1583-1590.
[3] 中华医学会神经病学分会帕金森病及运动障碍学组. 中国帕金森病的诊断标准(2016版)[J]. 中华神经科杂志,2016,49(4):268-271.
[4] 王永炎,张伯礼. 中医脑病学[M]. 北京:人民卫生出版社,2007:719
[5] 马大勇,韩振蕴,周杰,等. 通络化痰胶囊治疗脑梗死恢复期痰瘀阻络证353例临床研究[J]. 中医杂志,2012,53(7):563-567.
[6] 赵碎巧,操银针,李信华,等. 通络化痰胶囊联合阿托伐他汀对急性脑梗死病人血小板聚集度及炎性因子水平的影响[J]. 中西医结合心脑血管病杂志,2016,14(7):685-687.
[7] 王兆义,孟昭阳. 他汀联合通络化痰胶囊对痰瘀阻络证冠心病患者相关危险因素的影响[J]. 医药卫生,2016(8):45.
[8] 富苏,范吉平. 通络化痰胶囊对脑缺血损伤大鼠神经细胞凋亡及缺血半暗带CytC、caspase-9和caspase-3表达的影响[J]. 中华中医药杂志,2013(5):1194-1198.
[9] 富苏,韩经丹,周杰,等. 通络化痰胶囊对脑缺血损伤大鼠神经细胞凋亡及Bcl-2,Bax的影响[J]. 中国实验方剂学杂志,2013,19(8):242-246.
[10] 富苏,范吉平,王新志. 通络化痰胶囊对H2O2所致的PC12细胞凋亡的保护作用及机制[J]. 中国实验方剂学杂志,2013,19(12):188-193.
[11] BENZIE IF,STRAIN JJ. The ferric reducing ability of plasma(FRAP) as a measure of “antioxidant power”: the FRAP assay[J]. Anal Biochem 1996;239(1):70-76.
[12] KHAN MS,TABREZ S,PRIYADARSHINI M,et al. Targeting Parkinson’s-tyrosine hydroxylase and oxidative stress as points of interventions[J]. CNS Neurol Disord Drug Targets,2012,11(4):369-380.
[13] MOLAEE N,MOSAYEBI G,PISHDADIAN A,et al. Evaluating the proliferation of human peripheral blood mononuclear cells using MTT assay[J]. Int J Basic Sci Med,2017, 2(1):25-28.
[14] HWANG O. Role of oxidative stress in Parkinson’s disease[J]. Exp Neurobiol 2013,22(1):11-17.
[15] LU C,WEI Y,HU R,et al. Transcranial direct current stimulation ameliorates behavioral deficits and reduces oxidative stress in 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine-induced mouse model of Parkinson’s disease[J]. Neuromodulation,2015,18(6):442-446.
[16] SALIM S. Oxidative stress and the central nervous system[J]. J Pharmacol Exp Ther,2017,360(1):201-205.
[17] JIANG T,SUN Q,CHEN S. Oxidative stress:a major pathogenesis and potential therapeutic target of antioxidative agents in Parkinson’s disease and Alzheimer’s disease[J]. Progress in Neurobiology,2016,147:1-19.
[18] KOPRICH J B,RESKE-NIELSEN C,MITHAL P,et al. Neuroinflammation mediated by IL-1β increases susceptibility of dopamine neurons to degeneration in an animal model of Parkinson’s disease[J]. J Neuroinflammation,2008,5(1):8.
[19] WU D,TSENG IJ,YUAN RY,et al. Memory consolidation and inducible nitric oxide synthase expression during different sleep stages in Parkinson disease[J]. Sleep Medicine,2014,15(1):116-120.
[20] HUO Y,RANGARAJAN P,LING EA,et al. Dexamethasone inhibits the Nox-dependent ROS production via suppression of MKP-1-dependent MAPK pathways in activated microglia[J]. BMC Neurosci,2011,12(1):49.
[21] LANGSTON JW,IRWIN I,LANGSTON EB,et al. 1-Methyl-4-phenylpyridinium ion (MPP+): identification of a met-abolite of MPTP,a toxin selective to the substantia nigra[J]. Neurosci Lett,1984,48(1):87-92.